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Transformed DLBCL routinely add CDKN2A deletions and MYC translocations or amplifications in addition to the genomic alterations previously existing in the first CLL, but absence SITUS JUDI MBL77 the popular mutations observed in Most important DLBCL indicating which they may well correspond to a different biological class.eighty Richter transformation also occurs in individuals addressed with BTK inhibitors. These tumors usually do not normally acquire BTK or PLCG2 mutations but, if these have been current in the first CLL, subclones may arise with further unbiased mutations.89,ninety

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